Thyroiditis–the inflammation of your thyroid gland
Did you know that Hashimoto’s, a well-known version of autoimmune disease, is just one of many kinds of Thyroiditis??
THYROIDITIS is is an umbrella term to describe several conditions concerning the inflammation, or the attack, of the thyroid gland, of which Hashimoto’s disease is just one.
Sometimes you will also get the umbrella diagnosis of Autoimmune thyroid disease (AITD). That just means an organ specific autoimmune problem, which can include Hashimoto’s and many of the below, as well as Graves disease.
Symptoms of thyroiditis can first fall in the hyper end of the range (anxiety, nervousness high heartrate or palpitations, heat intolerance, etc), then end up being the same as hypothyroidism, including fatigue, weight gain, concentration problems, swelling and more. When treatment is finally needed, it’s the same as for patients with general hypothyroidism–natural desiccated thyroid.
THYROIDITIS includes all the following:
Hashimoto’s Disease, aka Hashi’s, is the most common form of thyroiditis and may be the most common form of hypothyroidism. Antibodies are present, showing an attack on your thyroid, whether anti-TPO or anti-thyroglobulin. Cause is not always known, but autoimmune issues seems to run in families for those who end up with Hashi’s. You can read more about Hashi’s here.
This is considered a milder, less aggressive form of Hashimoto’s and often without antibodies, which is why an ultrasound might be needed to firmly diagnosis Seronegative. One can have a goiter. For some, this might be the same as De Quervain’s below.
This happens after the birth of a baby, is common, and may play a role in post-partum depression. Antibodies will be present and you may have no pain in the thyroid at all! It will start out with symptoms of hyperthyroidisim, including a fast heartrate, easy weight loss, feeling nervous, tired, and not liking heat. It will move into hypothyroidism, and some women will remain hypothyroid the rest of their lives, while others will have a remission. Cause is not always known.
Subacute thyroiditis, also called De Quervain’s Thyroiditis
This form of thyroiditis will involve inflammation and pain of the gland and a quick release of excess thyroid in your blood, but antibodies are not found. It’s uncommon, and some feel it’s caused by a virus of the thyroid, and can occur after one has had a upper respiratory tract viral infection. Pain of the thyroid is common and can be acute. Some have fever. Taking aspirin to reduce inflammation, plus bed rest, is recommended by doctors. Patients can recover from this.
This condition is like having both Hashi’s and Subacute thyroiditis, but does not usually have pain of the thyroid, i.e. “silent”. For some, it is recoverable within a few months, tho others may have lifelong hypo. Cause is generally unknown.
This can be caused by the continued use of lithium, as well as amiodarone, interferons and cytokines. Will continue as long as the medications are used. These drugs may not actually cause thyroiditis, but just plain ole hypothyroidism.
Radiation induced thyroiditis
This rare condition can occur after the use of radiation treatment to treat hyperthyroidism, or radiation used to treat head or neck cancer. Pain is common. It can result in lifelong hypothyroidism. No antibodies are present.
Also called Suppurative thyroiditis, this is caused by a bacterial or other infection, and can go away with treatment of the initial infection. No antibodies are present.
This is where one’s thyroid will contain dense fibrosis causing inflammation, and this may be more of a fibroid disease. With this, you can also have hypoparathyroidism, hoarseness from problems to your larynx, and a compression of your tracheal tube. Your thyroid will feel quite hard.
Ord’s thyroiditis / Atrophic thyroiditis
This form of thyroiditis refers to a reduced thyroid size, and is basically very similar to Hashimotos, i.e. one or both antibodies will be attacking the thyroid. It’s more common in European countries. Ord’s/Atrophic Thyroiditis patients tend to see their thyroid atrophy rather than form an enlarged goiter.
** To read specifically about Hashimoto’s disease, go here.
(Important note: STTM is an information-only site based on what many patients have reported or learned in their treatment. Please work with your doctor. This is not meant to replace that relationship or guidance, and you agree to that by reading this website. See the Disclaimer.)