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Thanks to Dr. Lowe for a very valuable call last night

Dr.JohnCLoweAnyone who listened to the Thyroid Patient Community Call last night will know that we were listening to a powerhouse of a man with an encyclopedia of valuable information: Dr. John C. Lowe.

He is the author of an exhaustive book on Fibromyalgia called The Metabolic Treatment of Fibromyalgia, which explains how the proper diagnosis and treatment of hypothyroidism is a good choice in the treatment of fibro. He also owns drlowe.com and the research website Thyroid Science.

I already knew he was great. He’s listed in the Acknowledgements of the Stop the Thyroid Madness book as one of a handful of medical professionals who played a huge role in educating and empowering patients all these years. He, like the others, was a springboard for patients to move forward in gaining far better information about thyroid treatment and everything else we have have learned over the years!

And he has agreed to come back, since we had many more questions to ask him! I’ll announce that when we secure a date.

Below are some highlights from our talk with Dr. Lowe:

  1. The use of high dose B-vitamins is very important for the thyroid patient, and even more so when you are feeling better in your treatment. Thyroid increases the production of protein, which drives energy, and you’ll need the B’s to help this higher energy production.
  2. Sadly, it’s economics which dominant science and and researching, and it’s all for marketing purposes instead of the pursuit of truth and the real health of patients.
  3. What drives your medical investigations? To gain wealth, or to help patients?
  4. 75% of the time, if a doctor uses the TSH to either diagnose hypo or treat it, he will be wrong.
  5. In the 1980’s, the top of the TSH range was 7.5.
  6. Abbott Labs, the makers of Synthroid, gives one million dollars in an unrestricted educational grant to the American Association of Clinical Endocrinologist (AACE) i.e. no wonder Endocrinologists have a love affair with Synthroid, a medication like other brands which has left the vast majority of us undertreated. You can read more details from Lowe on this subject here.
  7. Isocort, an OTC treatment for low cortisol, is protected by the 1994 Dietary Supplement Health and Education Act.
  8. HC, aka hydrocortisone, is water soluble. Isocort is fat soluble. People need bile so the lipase enzymes are able to work on it.
  9. There are no studies to compare Isocort to HC, and should be.
  10. If swallowing, take thyroid one hour before eating for best absorption. If meal will have a lot of fat, 3-4 hours before eating.
  11. Elderly people have less Hydrochloric Acid and do a worse job absorbing nutrients. (And so do undertreated hypo patients, reminded Diane) Betaine is a good supplement to help.
  12. Why it can be a bad idea to swallow thyroid with meals: we never know how many compounds are in the food that will bind it, like calcium and iron. T4 is the most badly absorbed.

To hear more, just click above and listen to the recorded call. Thank you for being you, Dr. John C. Lowe.

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icicles2BRRRRR. In the US and many other countries, we are having one of the coldest winters we’ve seen in a long time. And if you are out in it alot, you may need a tad more thyroid meds to keep yourself optimal.

Dr. Lowe is coming to our party–listen directly or ask him questions one-on-one

Dr.JohnCLoweThis coming Thursday, January 7th, Dr. John C. Lowe DC, PLLC will be joining me for the Talkshoe THYROID PATIENT COMMUNITY CALL and it’s one you don’t want to miss.

Dr. Lowe has been highly respected by thyroid patients for years for the excellent information he has disseminated about better thyroid treatment on his website drlowe.com. In fact, he’s one of a handful of doctors that I mention with gratitude in the Acknowledgements of the Stop the Thyroid Madness book. His website and wise understanding played an empowering and self-educating role in the early movement by patients for better thyroid treatment with desiccated thyroid and better labs. You can read his own written biography here.

Mark your calender and set your cell phone alarm: the Community Call starts at 9 pm Eastern, 8 pm Central, 7 pm Mountain and 6 pm Pacific. You can log on anytime within 15 minutes before the call starts and participate in the Chat box. That same box will be right there during the entire call.

The entire live audio chat with Janie and Dr. Lowe will come right on your computer, so remember to turn your audio up.

Additionally, those who log in can also choose to call in and ask Dr. Lowe direct question. Don’t miss this great opportunity!

The Stop the Thyroid Madness Talkshoe page: http://www.talkshoe.com/talkshoe/web/talkCast.jsp?masterId=62603&cmd=tc

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See the blog post below for a very insightful Guest Blog Post by Amy about her role as an Undercover Thyroid Advocate. Below that, you can read how I was wrong about what it was like to be thyroidless, and several great comments.

Reverse T3–do you have this problem in excess? Let’s talk!

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This blog post has been updated to the current day and time. Enjoy!

Most thyroid patients have heard about T4…the thyroid storage hormone, also called a pro-hormone. You’ll see it in literature as “thyroxine” or “l-thyroxine”–the latter as the name for a man-made T4.

And many know about T3…the active thyroid hormone which rids us of hypothyroid symptoms.

And as patients become more informed, they learn that the body not only converts T4 to T3 through what is called deodination, it also provides some of that T3 directly. That is an important distinction! The latter fact can be why thyroid patients report getting far better results with natural desiccated thyroid (NDT) like Naturethroid, NP Thyroid or other brands.

Patients might also learn that there are actually five thyroid hormones made in your body, which is also what’s found in NDT: T4, T3, T2, T1 and calcitonin.

Reverse T3

But in every individual, whether a thyroid patient or not, a thyroid can also convert T4 to the inactive RT3 (reverse T3). RT3 is an inactive thyroid hormone, as compared to T3 as the active thyroid hormone. And converting to RT3 is a natural and necessary process, even if there are consequences.(1) The body might convert T4 to RT3 as a way to clear out excess T4, or as a way to reduce your metabolic rate. It can happen if you go through any of the following:

  • surgery
  • a major physical accident
  • certain heart problems
  • intense chronic stress
  • restrictive low carbohydrate diets (2)
  • chronic inflammation

When Reverse T3 is a problem

Unfortunately, many thyroid patients make far too much RT3, as well, and patients with their open-minded doctors have been making cutting edge discoveries about this fact. Many patients have seen that their high levels of RT3 can be found with the following conditions:

  • high cortisol
  • low cortisol
  • low iron levels
  • possibly low B12
  • lyme disease
  • gluten intolerance or Celiac
  • other undiscovered and untreated underlying issues that can go hand-in-hand with being hypothyroid.

Why is a high level of RT3 is problem? That excess RT3 is making itself lazily comfortable on your thyroid cell receptor sites, preventing the active T3 thyroid hormone from doing its job on that same receptor to get you out of your hypothyroid state. It becomes akin to a clogged up drain to your organs and cells. So you stay hypo and symptomatic, in spite of seemingly “normal” other labwork.

The solution

Informed patients discovered they needed to discover and treat all the reasons contributing to their body converting to excess RT3.

Want to read more? All the below is based on patient experiences and wisdom to share and work with your doctor:

For those with the revised Stop the Thyroid Madness book, there is also more good detail in Chapter 12 called T3 is the Star of the Show, page 155, to continue your education. This is all good information to take into your doctor’s office.

JanieSignature SEIZE THE WISDOM

 

 

 

 

 

 

 

(1) http://press.endocrine.org/doi/abs/10.1210/jcem-41-6-1043

(2) http://press.endocrine.org/doi/abs/10.1210/jcem-42-1-197

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